Electrostatic complementarity of B-cell receptor CDR3s and TP53-mutant amino acids in breast cancer is associated with increased disease-free survival rates

Cell Mol Immunol. 2020 Jul;17(7):776-778. doi: 10.1038/s41423-019-0328-8. Epub 2019 Nov 15.

Authors

Juan F Arturo¹, Boris I Chobrutskiy¹, Michelle Yeagley¹, Dhruv N Patel¹, Shayan Falasiri¹, Jay S Patel¹, George Blanck^{2

3}

Affiliations

¹ Department of Molecular Medicine, Morsani College of Medicine, University of South Florida, Tampa, United States.
² Department of Molecular Medicine, Morsani College of Medicine, University of South Florida, Tampa, United States. gblanck@health.usf.edu.
³ Immunology Program, H. Lee Moffitt Cancer Center and Research Institute, Tampa, FL, 33612, United States. gblanck@health.usf.edu.

No abstract available

Publication types

Letter

MeSH terms

Amino Acids / genetics*
Breast Neoplasms / genetics*
Breast Neoplasms / immunology*
Complementarity Determining Regions / metabolism*
Disease-Free Survival
Female
Humans
Kaplan-Meier Estimate
Mutation / genetics*
Receptors, Antigen, B-Cell / metabolism*
Static Electricity*
Tumor Suppressor Protein p53 / genetics*

Substances

Amino Acids
Complementarity Determining Regions
Receptors, Antigen, B-Cell
Tumor Suppressor Protein p53